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Since nitric oxide (NO) is known as the main vasodilative substance, generated in endothelial cells from its precursor l-arginine by the enzymatic action of endothelial NO synthase (eNOS) (15), we wondered whether these amino acids could stimulate NO production

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Simultaneously, the preservation of SAM levels maintains cellular capacity for critical methylation reactions including histone modifications, DNA methylation, and protein methylation that regulate gene expression and protein function